Acute obstructive hydrocephalus after subarachnoid haemorrhage.

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Acute hydrocephalus after subarachnoid hemorrhage.

This issue of Stroke includes an important discussion of acute hydrocephalus after subarachnoid hemorrhage (SAH), contributed by a group of investigators from The Netherlands who have made several important recent contributions to our understanding of the pathophysiology of SAH. The data of Hasan et al, derived from a careful study of 473 consecutive patients with SAH admitted within 72 hours a...

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Corticosteroid therapy of experimental hydrocephalus after intraventricular-subarachnoid haemorrhage.

Symptomatic hydrocephalus after subarachnoid haemorrhage seems to result both from mechanical obstruction of arachnoid villi and basilar cisterns and from an inflammatory cellular reaction in the villi. Subarachnoid haemorrhage was induced in rabbits using whole blood injected through an implanted intraventricular needle. Control rabbits receiving intraventricular methyl prednisolone acetate bu...

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Acute hydrocephalus and cerebral perfusion after aneurysmal subarachnoid hemorrhage.

BACKGROUND AND PURPOSE Acute hydrocephalus after aneurysmal subarachnoid hemorrhage (SAH) may decrease cerebral perfusion by increasing intracranial pressure. We studied cerebral perfusion in patients with and without acute hydrocephalus after SAH. MATERIALS AND METHODS We performed noncontrast CT scans, CT perfusion (CTP), and CT angiography on admission in all patients with aneurysmal SAH. ...

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Management problems in acute hydrocephalus after subarachnoid hemorrhage.

In a consecutive series of 473 patients admitted within 72 hours after a subarachnoid hemorrhage, 91 (19%) had hydrocephalus on the initial computed tomogram. Consciousness was unimpaired in 25 of the 91 (28%). In 11 more patients acute hydrocephalus developed within 1 week after subarachnoid hemorrhage. Thirty-eight (8%) of all 473 patients subsequently showed clinical deterioration because of...

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Rapid and effective clearance of cell-free haemoglobin after subarachnoid haemorrhage (SAH) is important to prevent vasospasm and neurotoxicity and improve long-term outcome. Haemoglobin is avidly bound by haptoglobin, and the complex is cleared by CD163 expressed on the membrane surface of macrophages. We studied the kinetics of haemoglobin and haptoglobin in cerebrospinal fluid after SAH. We ...

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ژورنال

عنوان ژورنال: BMJ

سال: 1984

ISSN: 0959-8138,1468-5833

DOI: 10.1136/bmj.288.6427.1342